The Peter Attia Drive - #287 ‒ Lower back pain: causes, treatment, and prevention of lower back injuries and pain | Stuart McGill, Ph.D.

Episode Date: January 29, 2024

View the Show Notes Page for This Episode Become a Member to Receive Exclusive Content Sign Up to Receive Peter’s Weekly Newsletter Stuart McGill is a distinguished professor emeritus at the University of Waterloo and the chief scientific officer at Backfitpro Inc. where he specializes in evaluating complex cases of lower back pain from across the globe. In this episode, Stuart engages in a deep exploration of lower back pain, starting with the anatomy of the lower back, the workings of the spine, the pathophysiology of back pain, and areas of vulnerability. He challenges the concept of nonspecific back pain, emphasizing the importance of finding a causal relationship between injury and pain. Stuart highlights compelling case studies of the successful treatment of complex cases of lower back pain, reinforcing his conviction that nobody needs to suffer endlessly. He also covers the importance of strength and stability, shares his favorite exercises to prescribe to patients, and provides invaluable advice for maintaining a healthy spine. We discuss: Peter’s experience with debilitating back pain [3:30]; Anatomy of the back: spine, discs, facet joints, and common pain points [14:45]; Lower back injuries and pain: acute vs. chronic, impact of disc damage, microfractures, and more [24:45]; Why the majority of back injuries happen around the L4, L5, and S1 joints [31:00]; How the spine responds to forces like bending and loading, and how it adapts do different athletic activities [36:15]; The pathology of bulging discs [43:15]; The pathophysiology of Peter’s back pain, injuries from excessive loading, immune response to back injuries, muscle relaxers, and more [46:00]; The three most important exercises Stuart prescribes, how he assesses patients, and the importance of tailored exercises based on individual needs and body types [56:15]; The significance of strength and stability in preventing injuries and preserving longevity [1:08:15]; Stuart’s take on squats and deadlifting: potential risks, alternatives, and importance of correct movement patterns [1:19:30]; Helping patients with psychological trauma from lower back pain by empowering them with the understanding of the mechanical aspects of their pain [1:30:00]; Empowering patients through education and understanding of their pain through Stuart’s clinic and work through BackFitPro [1:39:00]; When surgical interventions may be appropriate, and “virtual surgery” as an alternative [1:46:45]; Weakness, nerve pain, and stenosis: treatments, surgical considerations, and more [1:55:30]; Tarlov cysts: treatment and surgical considerations [2:00:15]; The evolution of patient assessments and the limitations of MRI [2:02:15]; Pain relief related to stiffness and muscle bulk through training [2:07:00]; Advice for the young person on how to keep a healthy spine [2:14:15]; Resources for individuals dealing with lower back pain [2:25:30]; and More. Connect With Peter on Twitter, Instagram, Facebook and YouTube

Hey everyone, welcome to the Drive Podcast. I'm your host, Peter Atia. This podcast, my website, and my weekly newsletter all focus on the goal of translating the science of longevity into something accessible for everyone. Our goal is to provide the best content in health and wellness, and we've established a great team of analysts to make this happen. It is extremely important to me to provide all of this content without relying on paid ads. To do this, our work is made entirely possible by our members, and in return, we offer exclusive member-only content and benefits above and beyond what is available for free.

If you want to take your knowledge of this space to the next level, it's our goal to ensure members get back much more than the price of the subscription. If you want to learn more about the benefits of our premium membership, head over to peteratea.com. My guest this week is Stuart McGill. Stuart holds the title of Professor Emeritus at the University of Waterloo, where he has dedicated 40 years of overseeing his laboratory and research clinic dedicated to advancing the understanding of back pain. Currently he serves as the Chief Scientific Officer of BackFit Pro, where he specializes in evaluating complex cases of lower back pain

from across the globe. He has authored 245 scientific articles and multiple textbooks. I wanted to have Stuart on for the obvious reason that very few people listening to this will have not had their lives impacted by lower back pain, even if it's just a short bout that lasts for only a few days. And sadly, many of you have had far greater impact resulting from lower back pain, lower back pain that has perhaps plagued you for many years. In this episode, we do a deep dive into all things that pertain to lower back pain. We begin by discussing the anatomy of the lower back and how the spine works, the pathophysiology of back pain, and where people can have issues as it relates to their back. We talk about why Stuart believes there is no such thing as non-specific back pain,

and why he is so adamant about finding a causal relationship between an injury and pain. And by injury, I mean a physical reason for the pain, not necessarily an acute injury that resulted in it. Talk about ultimately why people who are experiencing back pain should be empowered to do something about it. In other words, Stuart really believes that nobody should suffer endlessly because of back pain.

I'm very excited about this episode because I know even just looking at the relatively small sample population of my patients, I know this is a topic that many people will find value in. And if you're not finding value in it today, it might be a podcast you want to come back to when you do experience lower back pain, though I hope that never happens.

Lastly, this is an episode where Stewart shows off a variety of models and positions to better explain what we're covering in the conversation. So while the show notes will have all of the images, this may be an episode you want to watch on video. So without further delay, please enjoy my conversation with Stuart McGill. Hey Stuart, thank you so much for joining me today. Wish we were doing this in person because there's so much I'd love to get into, but I have a feeling we're going to be able to do a pretty good job remotely. And I get the sense that you're very well versed at communicating your ideas in two dimensions rather than three. So great to make your acquaintance today.

Same here, Peter. I've been looking forward to this day for quite a long time. At some point, I'm going to thank you for writing your book. You are one of the few people on this planet who, A, I allowed, and B, I did change my behavior. So thank you very much for that. Let's see where we go today. You're going to leave me hanging with that. I'm curious to know what it was. Were you a smoker who somehow stopped? No, I'm kidding. What was it? Well, a couple of years ago, my family doc right now is one of my former students. This may bring a smile to your face. I don't remember this, but apparently when he was an undergrad and he asked me to write the letter of recommendation for medical school, I told him, of course, I'm going to write this because one day I'm going to need a good doc when I'm an old man. Well, wouldn't you know? Anyway, so we did my blood and I was just on the edge of what the

cardiology association is saying, needing Crestor or Lipitor or something like that. And this doc knows me well enough. He said, let's run the experiment. We're doing it for three months. I'm living Peter Atiya's life. And then I love to work hard physically and finish it off with a beer, which of course, six days out of seven, I'm denying myself of that.

But long story short, I have my blood done again in two weeks and we'll see if this three-month experiment has paid off. He says, no, it's in your genetics. You're not going to move the marker. But my sister says, oh, no, you will. She did. Anyway, thanks and no thanks. But I think I'm sleeping a little bit better. I think I'm a little more mentally sharp, but we'll see over the next hour if that's true. We can revisit this while reserve the right to come back and say, maybe you don't have

to be quite as restrictive. I don't necessarily believe in denying all the pleasures of life, and I don't deny them myself. There were some paragraphs in your book that just burned into my memory that you allowed yourself some french fries and I thought, oh, okay, I'm going to stay with the plan, come hell or high walk. But anyway, there you go. I'm going to start with a story, Stuart.

It's a story that some of the listeners might know, but you probably don't know in this level of detail. It sets the stage for why this is a topic that is of great interest to me personally. Of course, by extension, I suspect that there are very few people who are going to listen to us today who can't relate to the subject at hand. The very abridged version of the story is I grew up doing all sorts of really aggressive things and really took to powerlifting when I was probably 14 and found myself reasonably strong

for a little scrawny kid. And between about the ages of 14 and 19, I really, really pushed, couldn't bench press to save my life, but seemed pretty strong in a squat and deadlift. And kind of ignored any claims my parents made that maybe I was doing a little too much. Truthfully and sadly had no formal instruction. I was just watching the other grown men in the gym who were insanely powerful and sort

of just trying to replicate what they were doing, but truthfully had no sense of what I was doing. Anyway, fast forward, I am 21 years old. I'm rowing at the time, so rowing crew. And for the first time in my life, I experienced lower back pain. This really rocked my world because I always thought that people who got lower back pain were people who did nothing. I never really thought someone who was as active as I was could get it. And for about two weeks, Stuart, it completely disabled me. I could sort of get around, but barely.

And being a college student, I didn't really have any resources. I didn't know what to do. This was actually, I think it occurred during the summer. So I didn't have classes, but I had to stop rowing. I remember that. And otherwise I was able to work. It went away and I thought everything was fine. And I never thought about it again, until the summer three years later,

when I was 24 years old, and I remember exactly where I was. I was in San Diego riding my bike up the steepest hill in San Diego, which is a certain patch of a mountain called Mount Soledad. There's a section of this thing where you make a sharp right turn,

and at that moment, it's about a 25 degree pitch. I experienced this very sudden pain in my lower back and like a typical idiot just kept on pushing and climbing to the top and finished my ride, but then went on to experience the exact same thing, Stuart. For two weeks, I was debilitated, couldn't do a thing other than sort of lay around and walk. But then it got better and I kind of just forgot all about it. And then fast forward to the big one. I'm doing tatter recognition here, Peter.

So the big one occurred in my third year of medical school. I'm now 27 years old and the remarkable consistency of this is not lost on me. It is every three years by the summer. The summer of 94, 97 and 2000. And I'm riding my bike from class to the gym. I get to the gym, hop off my bike to lock it up and all of a sudden I feel that same familiar horrible pain in my back. But this time it's a little worse than the previous two bouts. And it was so bad that I did something

I'd never done before, Stuart. I decided not to go into the gym. And so I just slowly got back on the bike and limped my way back to my apartment and wasn't able to do anything other than just sort of lay in bed. I assumed I'd be fine the next morning and I woke up the next morning and actually couldn't get out of bed. Luckily my roommate and I each had

separate phone lines so I was able to call him from my room. So began a really painful journey over the next couple of weeks where the only place I could find relief was bent at 90 degrees forward where I would basically stand and bend over the nurses station. By this point I was doing my clinical rotations and as every good gunning medical student knows there was no way I was going to miss a day of this. So I would drag myself into the hospital each day and somehow managed to get through this. The nurses took pity on me and so did the residents and they were injecting me full of tort all. And this went on for a month and it got so bad that eventually the pain progressed from just being debilitating in my lower back to a nerve pain that felt

like my foot was being skinned. And it was interesting in that the pain in my lower back to a nerve pain that felt like my foot was being skinned. And it was interesting in that the pain in my lower back started to subside as it was replaced by the feeling of my left foot being skinned from the bottom. I'm not going to go into the more details of the story because it gets worse and worse before getting better, but needless to say I have a graduate degree in back pain. There's a happy ending to this story, Stuart, which is after this bout, which occurred when I was 27, which took a year to resolve, I made it a mission to figure out what was going on.

I'm not suggesting that I have, but I know so much more now than I did then. Unfortunately, anytime I've had back pain since then, it has been a very, very short-lived experience. I'll plant one last seed before we jump into this just for both you and the listener so that we can come back to it. If you are to look at an MRI of my spine today, you would ask yourself, maybe not you,

because you're so well-versed, but a reasonable person would look at an MRI of my spine today at the age of 50 and say, how does he walk? This person must be in so much pain, he doesn't know his name. And yet I can tell you for the most part I'm not at all. Occasionally I get a little tight in my lower back musculature, but I don't have ridiculous pain. I'm not limited in anything I do. Again, suggesting that the correlation between the image of my back on an MRI and my symptoms is pretty light. So with all that as a backdrop, the fact that you're smiling so much as I tell you this story tells me not that you're taking pleasure in my pain, but rather the familiarity of my story. Exactly. I've been doing pattern recognition. There's only one thing that would account for

the repeated acute episodes. In the interim between each one, you were quite fine. Then it shifted to a ridiculous pain. And now you're at the stage of your life where it's more an occasional grumpiness when you cross what we call the tipping point. Did the pain go to your foot, Peter? Yes. Big toes or little toes? No, it was actually really interesting. It was burning pain that was like the bottom of the foot was being skinned.

I should have... There's one detail I should have shared with you that might explain this. When I finally did have surgery, it turned out I had a free fragment that was about five centimeters long from the L5S1 disc. So that free fragment had broken off. Well, I was going to guess this for you actually. I was going to ask you which foot. So the fifth root goes to your big toe. But anyway, you carry on. Yep. So basically, the really, really unbearable pain I was having, presumably, was because that free fragment was parked on the S1 nerve root. And even though it ended up taking two

surgeries to get that out, and those surgeries ended up causing more damage that needed more repair that turned into a journey of a thousand cuts. I was on the road to recovery, but the ridiculous pain seemed to be directly a result of the S1 nerve root. Well, if you want me to react to that story a little bit, I'm smiling because you told me exactly what the pain mechanism was. I knew it was a disc with an open-fisher disc bulge. It would be on the side of your foot, right or left. What foot was it? It was left.

Okay. So you had a posterior left-sided biased open-fisher disc bulge that would open and close as a function of the flexion posture, spending down to lock your bicycle, you just gave it to me every single time. And then you were able to vacuum that in. It lasted for a couple of weeks. Now you're in the unstable. Do you want me to show you a couple of mechanisms? What I was going to suggest,

even before we get into that, this is exactly where I want to go, Stuart, is let's walk people through the anatomy of the back. Now I understand that there are some people who are going to be listening to us, so whenever possible do your best imagining somebody can't see us. But I think there's also going to be enough people watching on video and will certainly refer people to the video, at least for this section, in addition to some diagrams. But let's really explain to people what this remarkable structure of the human back is.

The stability, the flexibility, the mobility, the amount of nerves, muscles, and ligaments that are involved. You could almost argue it's a miracle we don't get more injured, even though the frequency with which we do is intense. Take us through the anatomy. I would almost argue the opposite, Peter. There was a television show that they were producing and asking various experts around the world, if you got to re-engineer your particular area, me being the spine guy, and they had a cardiac person, endocrine system person,

how would you re-engineer it and make it better? And every expert said they couldn't. It was perfect. So everything in terms of systems in your body comes with a trade-off, and there are rules that manage the trade-off. So with that, I can start the anatomy. As fine as a series of vertebra, as you know, forming a flexible rod. This allows us to dance and move and procreate, tie our shoes and do all of these wonderful things. But at some point, you now say are picking your child out of the crib, you reach across the crib, gather your child, pull them in. If you had a flexible rod, consider a series of stacked oranges, it would fall apart.

So you need a flexible rod that you can then stiffen to bear load. You cannot push rope, but you can push stone, or in this case, an IV, to bear load. So all of these things are necessary to have a functional spine. What else can I say? Let's look at the structure of the discs, which are the fabric. The disc actually forms the subcategory of a biological fabric. It's not a ball and socket joint. Could you imagine if we had vertebra with ball and socket joints, you would need an enormous musculature around that flexible rod to control all the ball and sockets. You would need

an enormous motor cortex to coordinate all of these. You would be so wide you couldn't walk, you couldn't run, etc. But we have this very slender torso because we have discs. Now the stress-strain curve of a disc starts out with a little bit of a neutral zone in the neutral range. And as you approach the end range, the disc provides stiffness, a mechanical stop to motion. Fabulous. I didn't need all this complex musculature to do so. So the disc creates tremendous evolutionary efficiency in your spine.

Either end of the torso strategically is a ball and socket joint. The ball and socket joints of the hips and shoulders are designed to create power. Power is force times velocity. So if you were to watch a sprinter sprint, the extensor muscles explode like a hammer hitting a stone, a stiffened structure. If they hit rope, the hips would pulse and you couldn't run anywhere. You can't even walk without sufficient stiffness in the core. So I can get into an interesting discussion of how stability works approximately to unleash and enable this distal athleticism. So in terms of anatomy, we have a flexible disc that is a fabric. That great advantage is the efficiency of your dimensions

that I'm talking about, where light, narrow in the waist, we can run, et cetera. The price that you pay though, is being a structure of many collagen fibers. Let's take my shirt, which is a fabric. If I wanted to delaminate the fibers, I would have to create stress-strain reversals back and forth, and slowly we would debond the fibers. This is what happens to people's discs. They debond the fibers with too much load and motion simultaneously,

and this is what you must have done as a younger fellow. But the concentric rings of collagen that are held together with collagen type X binding substance, they hold a pressurized gel, which is this incompressible hydraulic fluid that creates the ball. That gets pressurized, but it's always seeking the weakness in the wall. If you delaminate the collagen fibers, then the nucleus seeps through. And in some situations, the fibers are pulled together and they create a fragment, as you described earlier. Or if it's an open fissure and contained underneath the posterior longitudinal ligament, there's a good chance it's going to get vacuumed back in and off you go for

another two or three years. I can talk about the nerves, I suppose. If you have a disc bulge, there is the spinal cord centrally behind the vertebra, and at each lumbar or spinal joint is a pair of nerve roots. Maybe one thing we can talk about before that, Stuart, is the other point of fixation, which are the facet joints. So if anteriorly this structure is bounded and the vertebral bodies are stuck together through their sharing of the disc, on the back, we have these other joints that come from each of them called these facet joints. So yeah, why don't you talk a little

bit about that? I don't know if you can see those, but the facet joints are guiding of motion. So you can see as I'm flexing and extending and twisting this model spine, these are articular joints in the back that are guiding motion. What you will find, I know what I'm going to find if I look at your MRI, at the level of the disc bulge, the facet joints will now be getting a little thicker, a bit more gnarly looking. Am I right? Because the Passette's almost always, two or three years after a major disc injury, they take much more load. Think of air in your

car tire. If you let a little air out of your car tire, it bulges on the road. It gets a bit sloppy to drive your car. You have to tune the pressure. This is exactly what happens with your body. So when you lose the controlling stiffness of the disc, you get more work performed on the facet joints and they wear a little bit faster than the adjacent joints and they grow thicker. And facet pain is very different from disc pain. It's more of a ache.

It comes on a bit more slowly. If you have a wound up facet joint, it can take two or three months to wind it down versus a disc that as you described, you can wind down in a couple of weeks. But if I can show this as a model now, this disc is normal. This bottom disc, L5 is normal, L4 has been damaged. I'm just going to apply a torque to this spine. Do you see how the majority of the motion now is occurring at the joint that's lost stiffness?

Think of it like a knee that has a damaged ACL ligament. It no longer has the guidance, and the rotation motion of the knee, which is normal, is now substitute with shearing motion. So shearing motion indicates it's the metric for instability. So now you can see the shearing instability and now look at the work being performed by the facet joints at the level of the disc being damaged and losing stiffness. Now those will get grumpy and they will wear a little bit faster if you continue with the behavior that you did prior to. So injury and this cascade changes the rules a little bit. So initially, the goal was to create power in the shoulders and the hips and transfer it through a controlled spine. But now the game has changed a little

bit. You're 50 years old, you will have a little bit of joint instability. It's more important now to create a muscular girdle around the joint that has lost a bit of stiffness. And for the next little while, do your core exercises, develop a bit more muscular control, arrest the shearing motions. And by the time you and I are very similar, by the way, so I'm in my late sixties now, similar by the way, so I'm in my late 60s now. My pain is gone. So the joint has become so stiff. I can still do everything I want to do, but the joint itself has stiffened up. Professor Ker-Caldi Willis, the famous Canadian spine surgeon, wrote a book called Managing Low Back Pain, and he described very well the process that most of us go through, the instability and the very acute episodes that come every two or three years that are very debilitating

to a muscular ache and you wake up in the morning on one side with this ache in your back, but if you push one heel away or put a pillow under your waist or something like that, you can get rid of the ache. And then if you live a little bit longer and behave by the new rules, I don't have any back pain. And I can encourage that you will seek that relief as well. Steve, give us a sense of the prevalence of acute lower back pain episodes. Is an acute lower back pain

episode defined as one that lasts up to some period of time, two weeks or something like that? No, I don't define it that way at all. You'll be surprised. I'm not the guy who can give you those statistics. I don't worry about those sorts of things. All I worry about is the people who come here and ask for help with their back pain. I'm not out there doing population studies to crack incidents. And even having said that, when I used to study that as a younger scientist, what is back pain? What's an acute episode?

Is it sufficient to be debilitating so you don't have to work? I was a professor. I could have an acute attack and go to work. If I was a construction worker, I couldn't. So even the definition of whether it was disabling or not gets lost. So I didn't really get into those statistics. But having said that, I don't categorize pain as being acute, lasting a certain period of time and chronic lasting a longer period of time because when we measure people here with back pain, very rarely do

we find chronic back pain. It's almost always due to them repeatedly insulting their back with many cute attacks and offenses all day long. So they think they have chronic pain because it lingers when we show them a strategy or whatever the treatment happens to be to stop the insults that occur throughout the day, all of a sudden their pain goes. And then they realized, you know, I never did have chronic back pain. So chronic back pain to us is pain that is intransigent, unrelenting. Their brains have changed. They've been traumatized. That is chronic pain and not always having a strong mechanical trigger. That's how we separate chronic and acute. But

the pattern that you described of the two-week disabling, terribly disabling pain you had, there's only one thing that that could be and that was an open-fishered disc bulge. So let's talk about the mechanism of the discomfort. For example, is that disc actually innervated? Is the pain that's being perceived due to sensory fibers of the disc? Or is it the response of the body sensing that damage going into some sort of protective mechanism that is seizing all the muscles within the proximity of it. Stretch. Or it could be both. So here's how I would answer that.

A healthy disc. By the way, all these models that I'm using, highly biofidelic models are made by dynamic disc designs. So when a disc is healthy, people say, well, what's the number one thing you can do to keep a healthy spine? And I will say, keep your end plates healthy. And they wonder about that. Don't damage your joints.

As you wrote in your book, if you damage your knee ligaments, you will now have in your last decade, disabled mobility. That's a fact. So it's the same with the spine. If you can look into the nucleus of this model, you'll see that there are red vessels and yellow nerves. Now, there are all kinds of papers, oh, there's no nerves inside the disc,

and then you'll read another paper, oh, there are nerves in the outer third, and then there are nerves all inside the disk. And then you'll read another paper, oh, there are nerves in the outer third. And then there are nerves all the way through. And the reason is a healthy virgin disc doesn't have any vascular tissues going into it, nor does it have any nerves. And the reason is, when you squeeze a disc, you build up tremendous intradiscal pressure that kills any kind of vascular sprouts or neural sprouts. It's a healthy environment containing the pressure. When you damage the disc and you lose the ability to contain the high pressure, now all of a sudden vascular sprouts grow in and so do nerves. So it's so unfair. You damage the disc and now the body grows a hardware,

more nerves, to feel pain even more. And then eventually this just goes to a very fibrous, gnarly structure, highly innervated, but now it just basically gristles to bone and all the pain goes away. But you can see where the damage line, if I can the contrast there, do you see those fibers posterior laterally on the right have delaminated? And if I squeeze the disc, then you see this I'm going to squeeze and flex. Do you see the fibers delaminating and allowing the nucleus to seep out? But here's the anti-dope peter, stay stacked and tall, and I'm going to squeeze the whole disc bulges in a diffuse bulging pattern, but nothing comes out of the delaminated region.

So there's a little bit of an explanation of why some studies will show an innervated disc and other shows they're not innervated at all. Think of where you get cadavers from. It's not young healthy people dying and donating their body. It's almost always older people. So those discs are innervated unless they're horribly down the cascade and they've grizzled and all the nerves have now disappeared once again. That's very helpful. And I was totally unaware of that, by the way. So that's very interesting. And as you pointed out, almost a very cruel adaptation that is quite counterintuitive. Let's talk a little bit about the curvature of the spine.

What is it about the way we interact with the world and the curvature of our spine that tends to produce the majority of injuries at either the interface between L4 and L5 or the interface between L5 and S1? Oh, what an interesting question. I'm thinking of several things that are going through my mind as you asked that. Well, first of all, it's the thickest part of the spine. So if I was to take a thin willow branch and bend the willow branch back and forth, no stress,

tissues damage because of one metric and it's strained. Not the force supplied, not the pressure. It's just strain on the tissue that is the metric of when it's going to disrupt. So it's thin, the radial distance to the neutral axis, which is the axis down the middle of that thin rod that doesn't go into compression or tension. It's all very low. Now let's take a thicker stick and we bend it and it shatters right away because it's much thicker. I'm gonna digress a moment, go back to the flexible willow branch. It's wonderful at bending, that's what it's made for,

but don't ask it to bear compression because it buckles right away. The thicker stick can bear tremendous compression, but it doesn't tolerate bending. So you look at the neck, very thin, small diameter vertebra. It's made for bending and mobility, fabulous. But as you move down the spine and get to the bottom two where the thickest is, they do not tolerate bending near as much as they tolerate compression. So there's the first bending near as much as they tolerate compression. So there's the first anatomic feature that describes why the bending stresses are greatest at the thicker two joints, which are at the bottom.

The other things that matter are the shape of the disc. So some discs are ovoid and the bigger the skeleton, they tend to go to a lima con. So you have the spinal cord there, and then the two lobes of the lima con. The bigger the spine, the more lima con the disc becomes. When you twist a lima con, you create a stress riser on the edge of each lobe. The bigger the person, you will see they don't tolerate sit-ups. Look at YouTube. Who is the man who has the world record for consecutive sit-ups? Do you think he has a thick spine or a thin spine? He won't be a powerlifter. Having worked with some fabulous powerlifters and strong men, competitors, not one of them does a sit-up. They train other things to tune their body and make it suitable

to that particular training stimulation. So now we see that shape, thickness determines why L4 and L5 are the target. We know that they don't twist as well as a slender spine. The facet joints are also very interesting as well. So some facet joints, since you brought those up earlier, are orientated like that in the sagittal plane. Others are orientated more open, as we say. So if you look at a gymnast who, by definition, me, I would never choose to be a gymnast. But you can tell, look at my facets, they're closed. I don't twist very well.

However, when you flex forward and pull a load, those facet joints just glide past one another. So a gymnast by definition is someone who has a lot of mobility in their spine. You will see that their facet joints tend to be open. Now, if I said to you, who among your patients gets spondylolisthesis? The broken pars bone that holds the facet joint on basically, you are going to say, oh, dancers, gymnasts,

the very people that had the mechanical advantage to twist. Now when they go into extension, their facet joints are like shingles on a roof. They bend the bar's bone creating stress strain reversals. And eventually that bone will get a stress fracture or a stress reaction and if they keep going full-blown spawn below this thesis. So there's all kinds of reasons. I'm just giving you a few now as to why those two discs really are the, as an engineer now, stress risers. When I developed, in my PhD thesis actually, a very detailed anatomical model

of the spine computer model, that hit home loud and clear. We did stress maps of real people moving. The pain and the injury was almost always at the site of the highest stress. And remember, I said, the metric is strain that actually leads to damage. Or it actually, if it's below the tipping point, it actually strengthens you. So we can have that conversation as well. What does not kill you makes you stronger.

There's a risk that you and I talking about this because we're both engineers will easily get into the weeds of compression strain, tension strain. But for people listening to us who might not have that background, can you explain the difference between stress and strain and what happens under tensile load, compressive loads, and things like that? Let's not talk about stress and strain. Let's talk about applied load and deformation. So stress and strain are normalized to an area. We won't get into that. If I apply a force to a structure, it deforms. I'm applying a force and I'm getting a deformation. A mature skeletal bone breaks at a certain amount of deformation.

A child's bone breaks at a different level of deformation. When you take a long bone and you bend it, the upper surface goes into tension. It's trying to pull apart. The lower surface goes into tension, it's trying to pull apart, the lower surface goes into compression. Some biological structures are stronger in tension than they are in compression. A child is actually weaker in compression than a bending bone, and then the adult is weaker on the tensile side. So a green stick fracture or a buckled bone in a young child would be very rare to see in an adult as an example. So the behavior of biomaterials when you load them and how they deform explains a lot of injuries.

So if you were to put me on the witness stand as people do occasionally to explain, explain is the damage that we see, professor, in this MRI or in the cadaver or whatever, consistent with this particular mechanical alleged scenario. Yes or no. And that's how we reconstruct that. Tissues, stress and strain, shear, bend, tensile pull apart, et cetera. And the deformation causes very specific types of damage. I'd like to use this example for people. I'd like to use the example of concrete, which is every engineering student's favorite example, right? So concrete is so strong in compression.

And yet, in tension, it is so weak that we need to come up with a hack. How can we use this material to allow it to be both strong in compression and tension? Because the example you use is really a good one. If you have a bridge made out of concrete and you're driving on top of it, the bridge wants to deform,

which means you're putting the top in compression, which it can handle, the bottom in compression, which can handle the bottom intention, which you can't. So we put rebar in because the steel rebar is of course strong intention. The saying is the whole purpose of concrete is to hold the rebar in place. When you think about the spine, I want to dig into this a little bit more if you think it's helpful. So we take an axial load on the spine and as you pointed out, the cervical spine is not built for tolerating a big axial load. It's designed more to provide movement.

It's a joint for great flexibility. The lumbar spine for all the reasons you just explained is really designed around taking a large compressive load and it's in the process sacrificed the mobility we have in the neck. But now let's talk about load in the context of flexion and extension, where you now do have within the disc, it's not just pure compression, maybe just even explain to people, flexion is bending forward, extension is going back. Now, if you have an axial load in that position, which you could easily have if you're deadlifting something or squatting something, any given disc, especially in that

lower spine region, can be under compression and tension at the same time, correct? under compression and tension at the same time, correct? Absolutely. I have a little bit of a story on that, Peter. It's so interesting when, say, I'm asked to give a lecture to a group of radiologists, and they describe very well all the subcategories of disk bulges and disk deformations and that kind of thing. But they've never been taught what the applied load nor the adaptation was.

So let me paint a little picture here of the deadlifter. A deadlifter almost always gets a posterior disk bulge, as you may know. So a deadlifter is under tremendous compressive load, and if they, say, get to the bottom of where the hips run out of room, now the femur collides with the pelvis and thereafter the rotation takes place in their low back. Because the nucleus is under such enormous compressive pressure, remember this model, I had to bend it forward to get the nucleus to squirt back. So you're creating a center of hydraulic effort post-degree. Now let's consider a person who's adapted their spine to do yoga.

This is why I say, please never mix up deadlifts and yoga. lifts and yoga. If you adapt your spine to be very flexible, you adapt the Type X Collagen, holding the Type I and Type II, the heavy, grisly collagen, and then the elastic collagen, all those fibers together. A powerlifter wants them to be stiff and tough. They even wear an exoskeleton of a lifting suit to add even more stiffness and toughness. But the yoga master, that would be the kiss of death. They want nice, viable, flexible spines. They soften the ground substance holding the collagen together so when they bend forward

in contrast to the disc bolts going backwards, the front of the disc now buckles under compression. So when a power lifter, typically now, of course there are very odd cases that are the exceptions, the power lifter bends forward and crashes the disc bulge posteriorly, but when the yoga person or very flexible spine, when they yoga person, or very flexible spine, when they bend backwards, the collagen

under compression buckles. So one gets a disc bulge from extension and the other gets a disc bulge from flexion. Isn't that interesting? And it all depends on how they adapted their spine. But my final point in all of that is don't mix up the adaptation schedules. So if you want to be a powerlifter, train your hip mobility, shoulder mobility, but torso stiffness. Try not to throughout the day do a lot of bending versus the yoga master. Please stay away from the very heavy loads. What is the pathologic response to the anterior bulging of the disc? Because when you have that posterior bulge, we should have mentioned this earlier and I guess it's worth stating, the spinal cord stops quite high up.

The spinal cord does not run down the entire canal. It stops around L2. So for most of the people experiencing lower back pain, vis-a-vis a herniation, fortunately the herniated disc is not hitting your spinal cord. It is hitting the nerves that emanate from it. But again, there's so much real estate in that area. It's insane because you don't just have the nerve roots. You have the dorsal roots. You have all of these other tiny little nerves that are going to the facets and to the disc and to the vertebral bodies that's running musculature. And to your genitals of everything that's important, of course.

That's absolutely correct. And I learned that the very, very hard way. Yeah. Yeah. We could tell some stories if we weren't on the air. Tell me about the manifestation clinically of the anterior herniation in that very flexible person who's presumably greatly lacking in any spinal stability. Dr. John L. D. They probably won't be too much. They will go along with their merry life and

along with their merry life and be flexible. The anterior bulge is not as a rule picking up any nasty nerve root compressions. And on the grand scheme, Peter, it's probably a non-clinical issue for them. Until they wanted to lift, they were in an emergency situation now. They've come across a car wreck. Someone is in the car, but they don't get them out, the car is going to explode. So we will all be placed into these situations at some point in our life, and whether or not we have the physicality to deal with them is another issue. But anyway, that's the downside of that particular adaptation and lifestyle perhaps. Which of these types of injuries leaves a person more susceptible to the movement of the vertebral bodies in a slipped fashion, where we now get that spondyloth. Never remember which spondylah we're talking about.

I think we're now talking about spondylothesis when the vertebral body on top moves relative to the bottom, correct? Yeah, that's the interior Dr. David H. H. a priori judge and attribute one of those to the symptoms, we always go by the assessment. It could be either spine for sure. I want to back up for just a second to the story I opened with and just kind of dig in a little bit more to the pathophysiology. So that very, very first bout of back pain I had when I was 21 years old. Clearly the previous eight years or whatever, maybe seven years, eight years of really, really, really heavy lifting. Certainly the

technical knowledge I have today about how to do these things correctly was completely absent. If you had to guess, and this is purely speculation, what was the process that led to that injury on that day, that manifestation? You know, if I had had MRIs examining my spine every year starting at the age of 13 until that first real insult at age 21, what would you have seen? Well, I've done studies. Do you remember the NHL hockey strike a number of years ago? Yeah. That was 94, wasn't it?

It was whatever year it was. No, that was baseball. But anyway, okay, yeah. Well, whatever year it was, the younger players, they would go to Russia and whatnot and still make a salary. But the older veterans hung around and I saw quite a few of them. You know, my shoulder colleagues, see some britch shoulders, I end up seeing them for low backs. But it was a fabulous natural experiment, Peter,

because they brought their MRIs every year. So say they were 11 year veteran, I would look at their MRs from the first year, the second year, and then I would watch the cascade, and then I would say, what happened in the eighth year? Oh, that was the year I started with a trainer, and the trainer believed in doing astagrass squats with a heavy weight. Aha. Look, what happened to the spine? When was the last time you saw a hockey player doing astagrass squat in the NHL? In any case,

that was a wonderful experiment to give us insight into what you're describing. And then the second layer of evidence that I would add there is, I'm probably only of a handful of people in the world. We had a radiology suite in our cadaver lab, where we would take cadavers and apply very specific loading scenarios to it, and we would watch the cascade of damage over time. So both of those I'll put together and give an answer to what I expect I would have seen. So we would have seen a lovely young spine in 14-year-old Peter, I think you said you started, and then over time we would have seen delamination from the inside out. So you were accumulating the delamination, but on the outside it was still Christine.

Peter never knew. And the delamination would continue to progress layer upon concentric layer until that day when you were 21 or whatever and the last layer was breached and the nuclear gel extruded just a little bit. Now, when you were fertilized as an embryo or a blastocyst, I guess still at that case, around the end of the first month, that little flat plate rolled, it's called neuralation, as you know, to create your primitive spinal cord. On that day, your mother has not given you an immune system yet. Now it's fused up that nuclear gel has never seen the immune system yet.

The end plates are pristine, it's never seen your blood, which is where the immune system is active. So now you're 21. For the first time, that nuclear gel comes out and sees the blood immune environment. It kicks off a hell of an inflammatory response and you couldn't even move. It locked you up and that's how strong and powerful that was. Takes two weeks to subside. Now, here's the rub. I don't know if you've been following some of the recent

literature on anti-inflamm. I was going to ask you, would I have been better off if I had taken a prednisone taper or had some local anti-inflammatory therapy? Of course, none of these were at my disposal as a poor, dumb college kid. Of course not, but I can't tell you how much joy I'm having speaking with you because your logic is fantastic. And the answer is, it could have gone either way. The anti-inflammatory might have cleaned up

the immune response and given you faster resolution or what the recent literature is showing. There's a purpose for that inflammatory response. It brings in the immune system and all the macrophages, et cetera, And it starts eating up the extruded material. Now, that process can go one of two ways as well. It can wall off what's extruded. And I wish I knew you then because I bet I could have got you into just lay on your tummy and breathe. And that vacuums in, in fact, we did experiments, we would create partial disc herniations.

And then if you traction the spine and give a little bit of motion, all I do is wiggle your legs. You can vacuum in the disc bulge in a matter of two or three minutes and people will say you're dreaming. No, we've measured it in some types of subcategories. That's actually possible. The answer to the inflams is at least some of the more recent data is showing dispense with the anti-inflammatories. Let the inflammatory response give the patient

health for two weeks. It's the best medicine for them in the long term because it is helping to reduce the long-term discharge. Whether there's any basis to what I'm about to say, I don't know, but I will just say that anecdotally, these days when I have a flare-up, and again, to be clear, these are really, really minor, Stuart. They don't interfere with anything I do, other than if that were a day

when I was gonna lift a little heavier, I would back off. Even that I, given that I don't squat or deadlift or do any heavy stuff like that anymore, it's kind of a non-issue. But what I find to be the most efficacious is not any sort of anti-inflammatory, but a light muscle relaxant like a baclofen. So not a benzo or anything kind of sedating, but just something that allows the perispinus

muscles to sort of relax a little bit. And frankly, use that to allow me to do some deep breathing. And we're going to talk about the three most important exercises that you prescribe at some point today, I'm sure. So it's mostly just a vehicle to break the cycle of tension, but not the inflammation cycle. And truthfully, more of that is not because I'm familiar with the literature that you've just spoken of, but frankly, because there are downsides of taking prednisone as well, and we have to be mindful of those. And I don't want to suggest people shouldn't take prednisone, but one needs to be

circumspect about the frequency with which they do it. Here's where I think you are now to answer the first question. I will bet this is where you are now. You've got a little bit of micro movement in a shear mode. So this joint isn't translating as it should. It's lost a little bit of height and those are the things that are causing the low grade aches, not kicking off the heavy acute attacks that he used to have as a younger man. Now, test number one. I understand your brother has a farm up around here somewhere and you occasionally visit. If you want to spend an extra day, come on by the Gravenhurst and we'll have some fun. But anyway, what I would do with you is I would

get you to stand just as you are and I will bet you stand differently when you get out of that chair after doing this podcast for a bit versus of youess walking around. So there would be a full convene and an intelligent. And if I palpated your erector spinae, they would be active. And I would have to coach you to open up your hips a little bit, ears over your shoulders, shoulders over your hips. And now all of a sudden we've achieved that muscular relaxation that you're after. So next time, before you think you need to take the relaxant, humor me, lay on your tummy. Again, I don't know your spine well enough, but I would lay on

your tummy, maybe put your hands, palms up under your hips, maybe make a fist. Again, I don't know where you are, but we would find a nice little relaxation place and then I want you to melt into the table every time you exhale. Keep doing that. And tell me, A, if that doesn't remove the ache, and we will play with your hands to realign that little shearing micro movement. And then stand up. We might open up your hips a little bit with a so specific stretch and then you will monitor your back muscles and see if

you've shut them down. But then if I said poke your head forward, muscles on, we're in back muscles off. Soften your knees a little bit. Some people they will stand with a strategy of ramming their knees back into hard extension. Feel your erector spining. Maybe it's just simply jazz knees and soften your knees. In other words, those little postural cues, I have a sneaky suspicion and I've seen you enough moving on YouTube and whatnot that I bet we could hack our way around that. So there's our challenge. Let's see if we can do that without the med. You got yourself a deal. I will happily add an extra day to my next Toronto trip when I'm up at my brother's farm. I'm sure my brother will want to join as well. We'll take you up on that.

I'm sure my brother will want to join as well. We'll take you up on that. Let's talk about those three exercises, Stuart. There are three exercises. There's two of them that I've done consistently for quite some time. I really fancy them a bit. The third one, the bird dog, I only do occasionally. But let's go through the three of them. And just for the listener, we're going to link to videos of these. So you're going to do your best to explain them and provide the rationale for them. But ultimately, a demonstration will be forthcoming through videos we'll link to in the show notes. But this is kind of like your core nutrition. This is sort of the everybody should be doing this. You don't wait till you have back pain to do this. Is that safe to say?

No, it isn't. This is a bit of a myth and something that I've been fighting basically my whole career. Miguel Big Three. There are some people that are far too stiff and this is not the mechanism of their back pain and we don't need to go there. Have you ever seen the type of body build where they have a huge pneumatic cushion in front called a belly? If it slaps on their thighs, it's that pendulous of this. Do you ever see spine instability in that type of architecture? I don't. Those people have difficulty getting on and off the floor.

The big three is not for them. Again, the assessment always leads us to the solution. I need to have a discussion of what stability is in terms of creating resilience and performance. Then why are those particular exercises important and then how to do them? If I could follow that logic, Peter? Yeah, let's do it. And then the other thing, Stuart, if you want to throw it in there, do you want to talk about some of the hallmarks of your assessment wherever it fits into those three things? Take it away.

Yeah. All right. So remind me, we're going to talk about non-specific low back pain and how I think it's a myth and it doesn't exist. That will take us into the assessment. So let's go back to a basic discussion of stability. I might use an example of a backhoe. So a backhoe is a machine with a tractor and it has an arm on the back to dig earth. The first thing the operator does is put down the stabilizer bars to lock the tractor into the ground because if you don't do that, you can't pull earth, you just pull the machine

around. So what's the human equivalent of that? We live in a linkage, just like machinery. In other words, let's take the bench press muscle, pec major. Pec major originates on my rib cage, spans my ball and socket joint of the shoulder, and inserts on the humerus. So when I contract and shorten the pec major,

it flexes my arm. So if I wanting to do a push or a punch, there it is. That's on the distal side of the joint. Proximately that same muscle shortening collapses my rib cage towards my shoulder joint. So all I used was the muscle that spans the joint. That isn't a very effective push. All I'm doing is collapsing my own linkage, or as an engineer, we would say, well, we just created an energy leak. I'm now going to build proximal stiffness. I'm going to lock my core, create stiffness through my torso, which is proximal to the joint. So now when I contract the muscle, 100% of the motion is directed distally.

Now I've got my push. So what is the best most efficient way to create a proximal stiffness? We searched for years doing all kinds of tests of every abdominal exercise. You could think of back exercises, twisting, towel off presses, throwing things, et cetera. The three exercises that kept bubbling up to the top

in the criteria of sparing the spine while you're doing them, because these people are hurting. You don't have carte blanche to load up their spine. A guaranteed stability or proximal stiffness. And it was later in my career that we found there is a residual stiffness that occurs. So if you do the big three and you are an NFL football team, if you do the big three prior to practice,

you will run and cut just a little bit faster. So you're on the field, you run and you cut. The stiffer, the core, when the hips explode into external rotation, you're now creating a faster directional change. So what were the exercises? A modified curl-up, which remember, I'm now just going to start a little bit of an assessment. I'm going to take a patient, I'm going to have them sit on the stool and I say, do you have symptoms right now? And let humor me and let's say they don't. Now I'm going to say,

drop your chest down. Does that cause you to, oh yeah, my left toe is going numb and I've got back pain. Good. Bring your chin down and they might say that, it'll increase their pain or decrease it, but the point is that posture created their pain. If that is true, when they lay on their back and they imprinted their back into the floor doing a Pilates rollup, for example, that would be their specific pain trigger.

So it's not much of a therapeutic exercise, but we can say, put your hands under your low back as you're laying on the ground, lift your elbows, now hover up your head, neck and shoulders. And we're going to propel the abdominal contraction, breathe through first lips and allow the diaphragm to become the athlete inside this barrel. So that was the foundation of the modified curlup. Now, if the person has a rotator cuff issue

or we will hack it and make it tolerable. Then I would see people, well, let's take a dumbbell or a kettlebell and we're going to raise it up laterally in the frontal plane like this for the side of the core. That would trigger pain in a lot of people. I'll demonstrate all this if you want, but we could then do a side plank on the floor. The beauty of the side plank is only half the musculature is heavily challenged. The downside is heavily challenged. The upside is not. You've only got half the load on the spine.

Very spine sparing. We prescribe it on 10 second intervals. Why? We use the Russian training science to show you build endurance through repeated 10 second exposures, not getting tired to the point where you break foam, nor do you develop a neural fatigue and you get a much higher tolerable training level with this, what we call the

Russian descending pyramid. And then for the back muscles, look at the beauty of the bird dog where you extend one leg, the opposite arm, one half of my low back is active, one half of my upper back is active on the other side, we're developing a nice PNF pattern. We're creating stiffness and stability in the core. We're teaching the brain to disassociate ball and socket joint motion of the shoulders and hips with only half the spine load of say a Roman chair extension or something like that. So that bubble got to be a fabulous exercise. Then we did experiments where we would train people.

We would just have a single session exposure. We would measure the core stiffness prior to doing the big three. They do the big three on the Russian descending pyramid and then we would re-measure their torso stiffness. Peter, they were stiffer. And some of my muscle physiology colleagues said, well, you've added a turgidness to the muscle. I don't think so. I think the brain created a lasting neural stiffness. And in some people, it lasts about 20 minutes. Some people, it lasts longer. So you will see some patients who say, you know,

when I do the big three, I don't have pain for the next hour. Fabulous. What you're going to do is mid-morning, do a 12-minute, big three session, mid-afternoon, do a 12. So these are the little tricks and hacks to slowly wind a person down out of pain. That was the inside of the big three. Then we started to look at the performance side.

If you train a group of athletes versus graduate students, the typical university experiment. Not much difference was found in the athletes, but in the graduate students, we would see an increase in stiffness over a six-week training trial. Now, really interesting things started to happen. If you do isometric holds in the manner I've described, you punch harder. We took a group of Muay Thai athletes, and when they did the big three and we measured the punching impulse, it was greater after they trained for six weeks. When we did dynamic core exercises,

it increased the closing velocity. So the closing velocity is when you first get the first muscle pulse, boom, and then you relax closing velocity, and then you strike with the second pulse, boom, boom. The closing velocity was faster with dynamic core exercises, but the strike force, boom at the

end, was greater the isometric big three. Again talk about performance. I know you're a bit of a pugilist. I certainly study combat techniques. You know, if we were to take three styles, let's take Joe Frazier and you would see him just always on forward progression. But the punches came from his body weight behind him. He would create a beautiful thrust line straight, but his body rotated and he lent his weight into them and that was his footwork. It wasn't the greatest for getting hit because that means you get hit a lot.

Mike Tyson, different body type, very compact type of a body, but contrast his footwork. Oh, it was just beautiful. He would drop step, drop step, drop step, hook the liver, come back very quickly, hook, boom, and cross, and there was the knockout. Again, all coming from the hips, drop step, boom, you see it's all hips, you know this, and then all the, breaks all the rules, the lulley shuffle, and then he would turn, rotate, hang on to it, and then at the end, flick it out. Beautiful thrust line all through the stick and core. I can go through athlete after athlete.

I saw the other day I've never worked with Mick Jagger, but there is Mick Jagger doing the bird dog in his training. Usain Bolt, the fastest man on the planet does the bird dog. Reading extensor pulsing power into a stone. Or just to finish that off, Usain Bolt does bird dogs. Bird dogs are beneath people. Really? They should see what I see. Anyway, that was the end of that story. I was just going to add to it by saying, I think that what I've become interested in

as I've aged is looking at the greatest performers. There's no doubt that the best athletes have a remarkable natural talent that the rest of us don't have. I've measured it without question. Where I think people miss the talent, what they're missing is a big part of the talent is the natural stability. In other words, it's the force transmission without the energy leakage. And when I contrast really good athletes with myself and I examine my athletic past. What is clear to me is that in everything I have ever done, despite all of my hard efforts, my lack of natural ability and at the time coaching has meant that I have always suffered from an unbelievable amount of energy leakage.

Whatever I have done, whether it's been boxing, swimming, powerlifting, all of those things, there's such a chasm between me and the really good ones. And it's not due to hard work. I can promise you it is not due to effort. It is due to probably some combination of natural ability and coaching that has allowed the really good ones to do what you've demonstrated, which is a great punch begins in the back foot and it's transmitted through the hip and it goes into the opposite fist. It's just hard for people to understand how that through line of force can't lose anything along the way. The stories I could tell you about the number of athletes being detuned by their trainers and coaches violating this principle that you're describing, it's astounding to me.

Why are you getting them to do that? You just detuned their athleticism. I think where I want to go with this is most people listening to this are not going to lament the fact that they didn't run as fast as they could have when they were younger or that they didn't punch or swim with as much prowess as they could have. Where I think we should all care about this is that it's not just that the energy leakage costs you performance. It clearly does.

It's that it predisposes you to injury. And that's where I think we have to bring this back. When I exercise today, I don't care about the performance. I care about the preservation and longevity of my body for whatever number of years I have left. So this is really where I think stability matters. It's what are the exercises I need to be doing? What are the exercises my patient need to be doing? So that as we age and we walk up the flight of stairs or carry something heavy, we don't hurt ourselves because we don't have that core stability that can resist the deformation that's going to allow energy to seep out of the system.

Well said. A story was coming to mind as you were saying that. I'll be giving a lecture or teaching a class and I'll show some data from an elite athlete. And there will be therapists and clinicians in the room who say, we don't deal with elite athletes. We deal with the elderly or we deal with sick people. And I think, what are you thinking? I'm showing you what the human body

has the potential to do. And your arrogance won't allow you to learn what is possible. And I'm going to give you a very emotional, I hope I can get through this, a very emotional I hope I can get through this, a very emotional story to show the arrogance that exists among some of our colleagues. Occasionally, medical groups, a hospital or whatever, will ask, would you come out and assess three patients in our auditorium in front of all our medical staff? I was at this facility, it was in Europe. The first person was a rugby player, fair enough, and I had 20 minutes and declared what I thought was going on. The next one was a woman in her early 70s, clearly distraught. You could look at her posture, her character, she was defeated by the world. She came onto the stage and I said, can you tell me your story? She said a little

and I said, can you tell me your story? She said a little few sentences, and then she said, but the therapist says that I have to leave my home now. When I get off the toilet, I'm a bit unsteady and she's afraid I'm gonna fall on the floor. I can't get off the floor by myself and I'm just gonna lay there and no one will discover me. I have to leave my home."

She started to cry at this point, Peter. She said, what's going to happen to my cat and all this sort of stuff? And I said, really, would someone please bring me out of school? And this will be our simulated toilet. So an assistant brought her to school on to the stage. I said, okay, pretend that's the toilet. Have a seat. She turned and had no idea how to move and just sort of plopped and collapsed on the toilet. And then I'm just going to turn this down because I want you to see my lower body kinematics as we're moving here. And then I said, would you get up off the chair? And I can't remember whether she was wearing a skirt or pants. Pants I think it was, but nonetheless, knees together and she just sort of collapsed and I had to help her. She was going to collapse onto the floor. And so I said, I want you to humor me now. You're my mirror.

When I coach, I try and use minimum words. I said, do this with your hands. Put your knee cap between your thumb and your hands as you slide your hands down. Good. Now, I want you to be a leaning tower, leaning tower forward and backwards and play with the curve of your back. Do you have any pain now? She said, no. And I said, watch my shoulders. You're shrugged. I want you to anti shrug. She did that. Perfect. And now I said, pull your hands up your thighs by pulling your hips through don't lift with your back. Pull your hips through. Hey, she had it done in three repetitions. That was now her pattern. And I said, Okay, think of what we've just done and sit on the toilet.

And I said, whoops, spread your feet apart. And there she went, slithered her hands down, and then she put her knees together. And I said, now stand up. She was going right back to the incompetent movement that caused her inability and disability before. I said, spread your knees apart and pull your heels underneath you. Sniff some air, now lean forward and do what you now know how to do.

And she did a perfect squat. Do it again. And then by the third repetition, big smile came on her face. This is the emotional part. I said, what's up with you? She said, I don't believe my home. Do I? I said, no. Do you know many of those hard-baked surgeons and clinicians started to cry as well? For the first time, they realized all I did was teach her weightlifting 101. And remember how this story started with the arrogance of some of our colleagues who say,

I don't want to hear stories about elite athletes. I deal with old people or sick people. And that's why they continue to not have the skill set to help their people. All I did was learn from the best weightlifters of the world, people who know how to move load, learn what the efficiency was and turn it into a hack to change a person's life. Anyway, that's a pretty emotional story and I hope we do that quite often. I know you like cars. Why does Honda race F1 race cars? Well, they don't anymore, but when they did. And the reason was they learned about automotive technology and the gear shift change in your Honda Civic came from the F1 race track.

So that's why we work with Elite athletes so I can bring it down. And I love working with them, of course. But they just give it away free to us and yet some of our colleagues are just so closed off they don't want to hear about elite performance. That's an absolutely beautiful story, Stuart. And thank you for sharing that. It's a sadly common story too.

And to me, I think the saddest part of that story is how many of those patients don't get the chance to sit on the stage with you for 30 minutes and learn that movement. You've been around long enough that I'm sure you have a better sense of this, but I feel maybe optimistically that we are in a place now where people are starting to appreciate the importance of strength and stability and that we're less afraid of this. There's more discussion of the importance of resistance training and that it's not a young guy thing to do. It's an everybody thing to do. But given the arc of your career, am I being just sort of delusional or do you really think that we're in a coming of age here? The way you phrase questions are fabulous. What was going through my mind? I try and answer every question. What's the evidence and what's the application? The evidence at the university,

with all our first year students, one of their course courses they took was on just basic fitness evaluation, range of motion, strength, hand grip, VO2 max, some of these markers. And they would measure each other, and we kept the scores year after year of the incoming class. The students got terribly soft, and I can prove it based on that data, and we would graph it. Now whatever year was the year where the students had grown up with the personal computer, it was right at the very late 90s I think. All of a sudden we saw the incoming class fitness plummet. Then something happened. They were a soft

bunch for about five years and then then slowly, to your point, they started to come back. And so I think your perception is right on. It did go to a terrible state. However many years ago, that was 15 or 20 years ago, but it is coming back. Now, among our colleagues, and having said that, I think some of them are terribly misguided as well. You know, they think, oh, you're not a real woman because I heard this on social media

until you can deadlift twice your body weight. Well, wait a second. If they could come here and see the number of people who've been caused by overzealous trainers and going bonkers on deadlift magnitude. Let's talk a little bit about that because I have to tell you, Stuart, I'm a bit conflicted personally, and I'll explain why. I obviously have no desire to do anything that I deem stupid anymore. My days of gritting through painful anything are long over.

I know the difference between discomfort that is worth pushing through and pain that is not. But when I think about in particular squats and deadlifts, especially around the deadlift, in exercise I really, really enjoy where I feel conflicted. On the one hand, I feel like now that I'm so tuned in to how to do this movement correctly, it's a really wonderful audit for my stability system. I'm embarrassed to tell you how much I didn't know when I was deadlifting. At no point did I understand the importance of tension in the arms, intra-abdominal pressure,

the variability in foot pressure on the ground, like none of that stuff, right? It was just pure brute force stupidity. Today, as I know those things, it allows me to modulate force and to, on a good day, push the envelope a little bit in what I perceive is safe. So on the one hand, I think, yeah, I should be deadlifting my whole life. I don't need to deadlift 400 pounds anymore, but I should be deadlifting because it's this great audit. And on the days that I don't feel that I back off. And then on the other days, I say, Peter, you don't need to do this anymore, because honestly, you can still get the same or nearly the same activation for all of the muscles involved using

other movements, single leg movements in particular, where you don't have a fraction of the axial loading. And yeah, you might need to do two exercises instead of one, but at the end of the day, there's a lower risk approach to get it. In other words, deadlifting is valuable, but you have a narrow operating window in which you can potentially hurt yourself.

So I continue to go back and forth on this, Stuart, as such. Here I am telling you, I still will go periods of my life where I'll deadlift every week and then I'll take three months off feeling like I don't want to push it. How would you advise a middle-aged person or even a non- middle-aged person who's thinking through this particular issue. Again, I have so many thoughts going through my mind. It's interesting when we have a back pain 50-year-old coming here and I'll say, what are your goals?

Oh, I want to set a personal best in deadlift. And I said, really? Okay. Let me tell you some stories. Let's talk about Ed Kohn. Do you know Ed Kohn? I sure do. The greatest powerlifter of all time. I was with Ed a couple of weeks ago. I'll tell you a funny story about him if you like in a minute. But anyway, Ed, when he would set a personal best, he'd take a

couple of months off afterwards. To set a personal best is so demanding of your body. There are actually, if you set a true personal best, most people experience micro fracturing just underneath the end plate of the trabecular bone. If you look at the great strength athletes, they train deadlift. And again, if you go to our website, look at the testimonials at the bottom, the number of world-class deadlifters who are on there. So I've worked with quite

a few of these people through their injuries. Now, those micro fractures could be a good thing or a bad thing. The professional powerlifter will take a week off. They train heavy deadlifts or squats once a week because it takes a week for the bone callus to not only attach through the chemical electro attraction, but to really scaffold on. It takes a week. If you deadlift in another three or four days, the way some trainers, they might deadlift a client three times a week. That allows those micro fractures to accumulate until finally you've got a full-blown end plate fracture or whatnot. So these are the

people that come here. And then I say, how about this for a goal? Do you have kids? Yeah, do you have grandkids? Yeah. How about this? I've since learned about your centurion Decathlon, I've since learned about your centurion, the cathalon, which I love by the way. I'll say, would you rather, as your goal, have the ability to play with your grandchildren on the floor when you're 80 and get off the floor and pick them up? And they pause for a minute and they'll say,

yeah, I like that goal. I say, well, you can't have both. If you think you're gonna continue having deadlift personal bests, you will have artificial hips and all of these other things because how many old powerlifters do you know? Do you really want to be like that group of athletes? So I can talk them into changing their long-term goals. Now is the time to get on the program and make sure you get there.

If that's the case, we eliminate deadlifts. We had an athlete here yesterday. They're at the end of their career. And I took them out and we went for a 10-minute walk to a hill that we have. And I'll say, here's why you're not gonna do deadlifts, but here's what I want you to do. I showed them a monster walk. Okay, monster walk.

Now, we're going to the bottom of the hill and I want you to lean back into the hill and we're walking backwards. You're gonna align your foot, ankle, knee and hip and push through the knee, through the knee, through the knee backwards through the knee, through the knee, backwards up the hill. You know, after 30 meters, they were absolutely done. Here they are doing all this deadlifting and they don't even have the leg strength endurance to walk backwards 30 meters. Totally inappropriate

stimulation of their athleticism to make it through 280. It It's a good one. For you. Let's do it again. We walked down the hill, we did three sets, they could hardly walk, and then we played the neurological grip, which I like to do a lot of. Now I said, walk forwards up the hill, but pretend you have $100 in your butt cheeks. Don't let anyone take it. Now walk fours up the hills and they say, I've never felt this before. The brain perceives exhausted quads. It now has to go and get the glutes. It's the only thing left.

So quite often we'll do an exhaustion focus to stimulate the thing that we really want to stimulate. And I convinced that person after that, what they're going to do and train now to get a well-rounded and sustainable athleticism that will spare their joints, still have great training capacity, but I think their athleticism

is gonna go through the roof. I've taken some very accomplished hour lifters, and we've taken out all the squats and just do sled work, backwards walking up hills. Some of these old time techniques, their joints settle down, they get a sustainable fitness, they lose this idea of maximum effort, squats and deads. And now they're thinking of the word sufficient strength,

sufficient mobility, sufficient endurance. And we've been doing this long enough now that we've tracked them and those are the ones that are getting through. Let's go get any one of our colleagues who are orthopedic surgeons. Tell us who you're replacing the hips of. Well, 50-year-old Caucasian women who have done yoga for 30 years, okay? Men around 50 who've done deadlifts over life. Who are you not? The middle of the road moderates, not the ones who've

rusted out and not the ones who've worn out, but the ones in the middle are the ones who are. So this idea of sufficient fitness, because I still believe we are all called upon to do things in life at certain times, I hope we're already enable. It's more fun too, just to be able to continue to do those things. So I'm like you, I don't do deadlifts, but I pick up 100 pound bucked up logs as an example, big oak log. So that's my stone lift. Load that into the log splitter.

Still split my wood. People comment on my hands. This athlete who came yesterday, I shook his hand when he came to the door. He couldn't fit his hand around mine. He said, whoa. When we were young, we didn't have dumbbells. My dad would give us a cinder block, cinder blocks. Anyway, as you know, the importance of grip strength, I will take any day over how much you deadlift. People often ask me, Stuart, why do you think grip strength

is such a great proxy for longevity? And I say it's the same reason I think VO2 max is a great proxy for longevity. Those are probably the two best biomarkers we have. It sounds crazy, by the way, that your VO2 max and your grip strength are better predictors of how long you're gonna live than whether or not you smoke, drink,

what your family history is for cancer. Like those things all matter, but it's amazing how dwarfed they are by those two. My best explanation for it is that those are the best two integrators for the work you've done. You can't cram for a VO2 Max the week before. If you have a high VO2 Max, you have done the work to get it. If you have a strong grip, you didn't just buy little grip squeezers on Amazon and filter away at them while you were on calls on Zoom. You had to do the work. You had to be

carrying heavy things, whatever it be, chopping wood, carrying cinder blocks, doing farmer carries. And of course, that also speaks to stability. That speaks to the stability that you have to be able to transmit force from the torso right to the hand. So agree completely. Let's pivot for a moment to talk a little bit about the amount of psychological trauma that exists in the patient with lower back pain. And I'm thinking very specifically even about some of my own patients or friends who have been in the throes of lower back pain. And if nothing else, Stuart, I take a great degree of comfort from the injury, the third injury that I had, the one in 2000, because it lasted so long

and because it was so debilitating and because I'm here today without pain. My confidence around small recurrences is so high that I don't tend to awfulize about it and work myself up. But I have great empathy for a person who doesn't have that knowledge and instead I don't know how to help someone sometimes because I can't tell what is mind and what is body at this point and I suspect that there's a significant interplay. So can you speak more about help someone sometimes because I can't tell what is mind and what is body at this point. And I suspect that there's a significant interplay.

So can you speak more about this phenomenon and what those of us who want to help these patients can do? I am certainly much more conscious of the point you're making now than I was 30 years ago, Absolutely. I'm going to start with a little story. This happens very often. You mentioned earlier how MRIs don't show you the mechanism of pain. Then I can give all kinds of reasons why. But let's take this patient. This is true. He came to see me. He said,

Hi doc, I hear you're different. I've got this pain. I've been everywhere. I went to the pain climate. They gave me narcotics and now they say the pain is in my head. I can live with the physical pain. I cannot live with someone telling me the pain is in my head, because that means I'm crazy. And if I'm crazy, I don't deserve to live. You've got two weeks. And in two weeks, I'm blowing my brains out. Now, there's a heavy psychosocial challenge. And a little bit of a story of what the system does to people. And it's not unusual for someone to come here suicidal. So I said, all right, you don't appear to have pain right now.

And he says, no, I don't. And I said, okay, what causes your pain? And he said, well, it's when I do a certain movement that I get a flash of pain and it feels like someone has broken a beer bottle and have ripped open my hamstring muscles. It's awful. And I said, oh, can you show me the pain? And he said, what? You want me to show you how I create the pain?

And I said, it's the only chance I have to understand it. I said, you've been to 15 different clinicians. Has no one ever asked you to show them the mechanism of your pain? Has anyone ever touched you? He says, no. I said, well, it's the only way I know. Peter, I put on my instrumentation, which was muscle EMG over the torso, the glutes, et cetera.

We put on the spine motion monitor, 3D motion spine monitor. And then I said, all right, let's see what causes this. So he stood there and he did a very weird thing. And he said, all right, well, here you go. And he wound himself around in a circle like this. And when he got to 10, top dead center. Now at that time, I heard like a little cavitation, little pop come out of his back. And that was the trap of the sciatic nerve. And he was in a bad way. I laid him thrown on a table, tried to give him a bit of decompression, and he went home and I said, I know exactly what the mechanism of your pain is. Here's what you should do over the

next three days, but I want you to come back, but promise me you aren't gonna do anything silly. Remember what the threat was hanging over us. He said, I promise. I called him that night, I called him the next day just to make sure. Then he came back and I said, I know exactly what your mechanism is.

Here's what the data showed. As he was winding himself around, he was using muscle. Muscle is stiffening and stabilizing. It's centering of the joints. And as he got to top dead center, he shut all his muscles off. He completely relaxed. And then there was a little shear translation or a clunk. And that's what we heard. And that's what scrapped the sciatic route. I said, okay, you have no pain. Push my fingers out harder. Good, hold that. Now talk to me and keep talking to me with that controlling.

We coached them through this in a minute, very simple. Keep the tone now and we're going through. And as he came to top dead center, you could see him. Ah, ah, ah, ah, ah, ah, I said, we're going through, and as he came to top dead center, you could see him, ah, ah, ah, ah, ah, ah. I said, we're there, do it again, hold on, keep control. He didn't clunk. Now it took him about four months to wind down the ache, but he never had another clunk or a trap.

10 years later, he brought his daughter to me. I saw her for back pain and he brought me a case of beer. I said, I did my one year follow up with you, but how have you been? He says, fabulous. I said, did you ever get another episode? Never had one. Now, some people will think that that's

a fantastic impossible story. Pete, after that one coach class, when he gave him, he was so coachable and he got it. He understood, he was a mechanical mind. He never had another acute episode ever. So, a suicide case from the medical system, not having a sufficient evaluation procedure to really get at what the mechanism of his pain was to a point where they defaulted and said, we've tried everything with you, it's not working. Therefore, the pain is in your head. The key was to prove to him immediately that he had the ability.

It's just he had to be shown how. So it was a process of understanding the mechanism, giving him a strategy to address the mechanism, and the psyche just changes. It empowered him. May I give you one more story? Absolutely. Okay. I was giving a lecture in England, and there was a fella off to the side and he was slumped down. Now, if you get a clinical psychology textbook, the picture of depression is this, knees together, slumped down in that demeanor. Now, if you have a posterior disc bulge, that is not a good position to be. So there we're starting with clinical depression, beating a discolch. So don't go together. And he just sat there. And then in the break, he came over to me, very quiet, spoken fella. And he said, I hear what you're saying. Do you have 30 seconds for me to tell you

my story? And I said, sure. He said, I used to be a police officer, hurt my back. I went through the NHS system. They only gave me exercises that hurt me more. Finally, they gave me a pamphlet, how to live with your back pain. And he said, that book destroyed me. What, you mean I have to live the rest of my life with my back pain and no one's ever touched me or shown me any of this?

And I said, oh, and then you'll remember that spot procedure that we went with the older woman that I described earlier. I simply showed him that. And he went back and he sat down on the chair, nice and tall. And then at the end of the lecture, I went over to him and I said, how's your pain? And he stood up and he said, it's gone. And he started to cry because he realized now what the system had done to him. In the meantime he lost his job and he realized that he'd been stolen from and those are his words. He said they stole my career from me giving me that book how to live with my backpain. Why didn't anyone show me what my pain was like? You just did in 30 seconds. I've been watching this pattern

for so many years you could see it a mile away. Anyway, those are two stories to link the mechanics and ultimately what we're trying to do is to empower people in showing them they have the ability within themselves they just need to understand the mechanism. And most of the time, they are able to mitigate the cause and then build a robust foundation. So I wrote back mechanic, and I started the experimental research clinic at the University of Waterloo. Maybe you've heard of this, but I've never heard of another clinic where they follow up with every single patient that they ever saw. We did a two-year follow-up with every single patient who came in and we subcategorized them because we assessed everyone into the mechanism of their pain pathway.

into the mechanism of their pain pathway. We gave them an appropriate exercise prescription. We followed up to see, did they even comply because some people didn't? And then how are you doing after two years? If you were in the subcategory that everything has failed, you've been told you need surgery. So you're at the end of the road now, you're a surgery case. In the two-year follow-up, following the plan that I just described for you with this thing

called virtual surgery, which is part of it, 95% reported that they avoided surgery and they were glad that they did. So that's my efficacy to the empowerment and psychology issue. Marshall What stands out to me the most in those stories, Stuart, is your consistent adamant drive towards understanding the mechanism of the pain. So it's, how do we break this down into a physics and biology problem? And I guess my question is, which type of healthcare providers are most in line with that? Is your PhD through the School of Kinesiology? Yes. I should back that up. Yeah, there's a lot of mechanical engineering in there, but

nonetheless, yeah, basically. But when we think of all the different practitioners that interact with patients who have lower back pain, ranging from neurosurgeons, orthopedic surgeons, chiropractors, physical therapists, kinesiologists. I mean, there are so many people and I never want to suggest that the profession determines the school of thought. Like I really think there are great people and there are lousy people within all of those categories. But what are the characteristics that you see driving that type of search for a true mechanistic understanding of the pain because I'll be honest with you like in all of my back bouts of misery

Nobody ever explained to me what was going on. I mean nobody said to me this is happening even as a medical student Yes, I could look at the MRI. I could see the fragment It clearly had to come out presumably, given that I was in such excruciating pain and the thing wasn't, you know, it might have taken months for the thing to have been resorbed. But there wasn't a sort of, we need to understand the why this is happening, so that we're going to fix the underlying behavior that's causing it. That's the thing that strikes me as the most interesting of those stories. And I guess what my long-winded apologies question is, is that a function of the individual

or of the school of training? Both. So the elephant in the room here is there is no billing code that exists for an assessment of back injury mechanism. Doesn't exist. You can't bill an insurance company and say, well, I assessed the person's back pain. When I started the experimental research clinic, I set aside two hours to see a back pained person. And that guy is all I ever saw, two hours. My medical colleagues who'd been through medical school training, which I had not. I'd only have ever been a guest professor

at a medical school, but I sure didn't graduate from one. My medical colleagues said, two hours, what are you gonna do for two hours? Well, I've been spending 30 years figuring out how I'm going to test sheer tolerance to compression, pulling a nerve root one way, pulling it the other way. Is it flossing? Is it friction? Does it stuck? Et cetera. Again, I set up a handful of people in the world that would take cadaverics, finds and create the injuries. I knew how to

measure them and what to look for in terms of the full pattern. But that's the first to look for in terms of the full pattern. But that's the first political impediment to all of this. There's no billing code. Therefore, you're left with clinicians who are billing for a procedure that they've been trained to perform. Well, if you have nonspecific back pain, it's an absolute crapshoot, whether a manipulation for mobility, an exercise prescription for stability, just a movement tool, not to create a stress riser or a stress concentration on the tissue that is sensitized, simple as that. So where I've arrived at with all of this, we have to train our own clinicians. And that's what I've been doing through Backfit Pro. And I do not care if you come from a chiropractic physical therapy, coaching, training, physiatry, neurology, radiology even, Background, all I care is that you have passion. It's a 50 hour online course of me going through anatomy, physiology, neurology, psychology, biomechanics, etc. And then the probably 100 subcategories of pain mechanisms, and then how do you test for all of these,

and then how do you coach them? And then after all of that, we have three days together where we do hands-on skills training at a table. So again, there's no subcategory in the medical rubric that explains how to assess back pain from the perspective of biomechanics, psychology, neurology, physiology, etc. They don't exist. So that was my challenge. Stuart, what's the name of that course? It's called the Summit Course and you can read about it on backfitbro.com. And is it only for practitioners or is there a variant of that course that an individual can

take to become sort of the master of their own domain? Okay, good question. It's mostly for clinicians. It's only been clinicians that I know of that have ever registered for it. I don't think we would stop a member of the lay public because some of them are very savvy from taking it. However, the gatekeeper of all of this is there's a fairly extensive exam at the end. It is a written exam. There's a practical exam where the person must assess

a real patient, usually online, with one of our examiners. They have to come up with a written explanation of the pain pathway and then a program of what they're going to do with the person, and then they have to coach elements of it. So they have to see the coaching skill as well. So that's sort of a gatekeeper at the end that I think would only be for clinicians. But that's the only way that I've found possible. I'm like you, I'm very agnostic in terms of preparation. There are fabulous chiropractors and there's the absolute opposite. There's fabulous therapists. There were fabulous professors and terrible professors. It's just the way it is.

Dr. Darrell Bock It's a very interesting course. It's almost something I wonder... I'd love to figure out a way to make the time. So it's 50 hours online plus three days in person is what it sounds like. Correct. Yeah. Let's talk about the cases where you think surgery is really the best course of action. And again, I think it should always be stated that surgery without understanding how you got there and then making sure you correct it post-surgically is not what we're talking about. So it should always be assumed that you want to understand what got you there. But what are the indications in your mind for where a patient is better off getting

a surgical procedure and we could talk about what do you think are the best indications for dyskectomy, fusion, etc. versus where would you take a contrarian approach where many people would say yes, surgery and you would say let's push a little bit harder before. Wow. A lot of elements there so I'll just start at the beginning and hope I can create a logic story. I did mention the follow-up that we did where 95% of people who were told they needed surgery,

in fact, avoided it. And what we did there was I anointed them and said, there is your virtual surgery. This worked really well on people who I'll paint the picture of let's take a stay at home mom with two young kids, every day has to go to the gym and ride the elliptical for 20 minutes, do something else as a stress reliever. Otherwise she's gonna murder us. You've heard that story before. I'll say good, go get your surgery.

Are you gonna do that tomorrow? No, you are going to lay in bed. You're gonna behave like a post-surgical person. You're gonna get out of bed and go for a pee three times. That is your total workload tomorrow. And slowly you're gonna build yourself back. In other words, surgery may work for you because it's forced rest.

Now I'm gonna give you a tool that will mimic the forced rest. It's called virtual surgery. Tomorrow, here's forced rest. Now I'm going to give you a tool that will mimic the forced rest. It's called virtual surgery. Tomorrow, here's the plan, here's how you're going to behave. We are going to desensitize strategically the pain mechanism as we've measured it, and we're going to retune your body with strategic mobility and stability plus movement skill so we don't replicate the stress concentrations that caused your problem in the first place. Let's see how you are. Now, if they can do that, 95% will avoid surgery. So there's my first little story for people in that category.

Stuart, just to be clear, what are the patients who you would not offer that virtual surgery to? Give me an indication where you would say, you know what, this is too pressing. Right. Obvious red flags, which before we see a patient, we don't take patients off the street, never. They always come through physician referral. So I'm hoping they've been checked for red flags. Do you know how many have not? Even though we state in the referral directions to the referring medic, we've had cases of aortic aneurysm, lung embolism, cancerous tumors, metastasized, all sorts of things

that somehow these poor people got through the system, and we were the ones that found it and saved their lives. I wish that wasn't the case, but all of those obviously are surgery cases and they should never have come to us in the first place. So obvious red flags is number one. Number two is when the pattern doesn't fit. So I was smiling when you were telling your original story, only because it was such a familiar spot on pattern consistency. You fit the pattern. I knew exactly what it was. When the pattern doesn't fit, I'll say, no, something's not right. I need you to go back to your dock. And here's the reason why

there is a turgidness under your liver. We're not able to move that pain by moving stress concentrations around your spine. So it's not a nerve. It's nothing vertebral or facet. The pattern doesn't fit. It's something else. So there is a person where we refer back and say something needs further investigation. But now the last part of your question was about the need or when we would say for a person you're not our person, you need to see a surgeon. Surgeons, and by the way we see far too many post-surgical patients who they went through, maybe the surgery was botched.

When I see a horrible scar on the outside of the skin, I think, man, if that's the pride that the surgeon took on the outside, what carnage has gone on the inside? Or sometimes it's a shit happens story. The nerve scarred in and adhered, ah, that's rough. Or the post rehab was terrible. Here's a person, they went to a fabulous surgeon and the surgeon says, oh, go do PT, that's your rehab. And the PT goes and gives them toe touches or something after they've just had a microdisk surgery. And guess what? They're re-herniated again, and now we're seeing them. But when would we say, no, you're not for us? The surgeons are at their best in cases of a real heavy stenosis. So there's not much room in the neuro canal.

The facet joints are thick in behind. So you've got encroachment from behind. You've got a calcified disc bulge coming from the front. So a couple level laminectomy to give the nerve some space. That really is when the surgeons are at their best. Some of the spondyloemilopathies that we'll see in the neck, I think of a lead lawyer in the courtrooms. And the judges would ask him,

sir, are you drunk? And he said to us, well, when I stand, I start my presentation. I'm fine. He says, but after two or three minutes, I'm losing my balance and falling over. And the judges think he's drunk. And then we found it. It was a cervical spondylomyelopathy that was also co-presenting with back pain,

but no one had figured this out. So that was a surgery case, obviously. So it's either post trauma, and then that one's obvious. They need a little bit of hardware to stabilize their spine. But it may also be spondylolisthesis. The listhesis or the shear translation is just choking off the tataquina or another nerve. We recommend surgeons who we have really good luck with. And in that situation, if the spondylolisthesis is significant enough, is the only treatment

of fusion? I'm going to say yes. There's no amount of stability you can generate in the perispinous muscles, in the QL, in the psoas to compensate for that. I mean, I realize that you have to forgive me because I'm not an orthopedic surgeon, but I would assume that there's some threshold. One millimeter of spondylolisis might be tolerated and at some level they would say, no, it's too unstable.

I wouldn't agree with that, Peter. It's not the distance at all. You go with the assessment. Again, the evidence I offer there is we're coming down to the next Olympics now. So I don't know how many Olympians and people who are tapering now for the Olympic trials we've had here over the past year, but this is every four years we're inundated with these types of athletes and they come in pairs where we might have two young women who are competing

for a place on the US Olympic team. In gymnastics, both have the same spondy. One will say, we need six months off here of gymnastics, and here's what we're going to do. We're going to do a heavy stabilization program. The next one says, oh, no, we really got to make the trials. We're just going to keep going with going to gym. And I can almost predict with 100 percent accuracy who's going to make it. So I wouldn't say at all that we don't try a heavy exercise stability program, regardless of the amount of slippage. And I've done that with people trying to make the special

forces in the US. You got to do a speed set up test. You got to do all of these things. Oh, but you got to have these fondly. Okay, here is the program to try and get there. You might make it. What about nerve pain? What about patients who are either having weakness such as a foot drop or Significant pain like the pain I had we have them all the time If I can get the nerve pain to move on the assessment, please don't have surgery Let us have a try at it. Most of it. They will be pleased Wow, we have to play with certain rules Give me an example of some of those. So let's say your assessment comes out that this person who's having intermittent sciatic pain and you do an assessment and you say, look, there is no doubt that you have a ruptured

annulus here. You've got a protruding segment of disc and it is clearly at times, depending on your activity, getting nearer to the nerve root. It's driving that sciatic pain. But during your assessment, I assume what you're getting at is through some of those positional things such as laying the person on their front, manipulating the legs, getting the herniation to retreat into the annulus. So you're saying if you can demonstrate resolution under a changing movement pattern, that gives you enough confidence that this doesn't need to be removed surgically.

Not resolution. Can I move the pain a little bit? Can I make it worse? And can I make it better? Now I'm starting to understand the variables that make it worse, make it better, and I play with those. I'm trying not to sound boastful. I'm trying to be scientific here.

There was a day not that long ago, I'm losing track of time. It was probably, well, it was the NHL playoffs. So there's our time marker. I don't watch TV really, but for some dumb reason, it was Saturday, I flipped on the TV, was the NHL playoffs, and I listened to the announcer, the name, oh, that's my patient. Next player, my patient.

Two of my patients are now in the NHL Playoff series. A little bit later, I flip over to TSN, Tennis Tour. I look at that, my patient. And then that night, the UFC comes on, there's my patient again. So in one day, I see three different pro sports, every single one of them had sciatica when they came to me. That's some evidence that I can offer.

Now I remember one of those players in the NHL, if he fully flexed, he would stir up sciatica and increase the risk of a full blown accused attack, as you and I know very well. So we got him to move well. He played hockey mindful of a skating style that he didn't get too flexed up. We didn't allow him to tie his own skates. He said, tying my own skates really set my backup. I said, good. Now, NHL players are very particular how they tie their skates, but they coached one of the training staff to tie his skates for him. Now, I know some people will laugh at that, but that was all part of the plan to keep the capacity as high as he could to utilize in the game. How he sat on the bench was also instructed.

The fellow in the UFC, this is no slouch, Jiu Jitsu really put his spine in a place where it could fire off an acute attack. You do not want to be in the cage fighting for your life and having an acute attack. That's the last thing you want. We would limit the mat time on Jujitsu. He would do stand up, all kinds of things to minimize the accumulative stress

on the disc bulge causing sciatica. He competed. I wish I could tell you who he was and what he did that night. So I'm not afraid of nerve irritation, sciatica, etc. And it certainly doesn't fall into the category of you need surgery. We've proven that far too many times. But as I said, heavy instability and when we fail to arrest the shearing movements, trapping nerves, it's gone on for quite a time. We can't hack our way around it. It's best to see a surgeon, a stenosis. Yeah, and stenosis as well.

Yeah. And it's many, many different forms. Central stenosis as well. And it's in any different form, central stenosis. It might be a foraminal stenosis and a bit of arthritic activity where they can just basically take a Dremel tool to describe it for your audience and burr out around the foramen or the whole, the lateral nerve comes out. Another one is, I know a lot of our medical colleagues say, well, a tarloff cyst, the neural cyst, well, they don't cause pain. Really? I will prove to you very quickly whether or not that's causing pain by pulling the nerve

root one way or the other. Typical pattern recognition might be a physio might do a slump test, which is you straighten one leg and you flex the spine and neck. But the net stress in the middle of the cord is zero. You're pulling it one way, you're pulling it the other way, it just goes into a little bit of tension. If that's a tar-off cyst, that won't be triggered. A tar-off cyst doesn't like being pulled one way.

So that patient on exam might say, well, I don't get pain with a slump test, but I can't stand driving my car. Oh, tell me about your car. Well, I sit upright, put my head back, and extend my leg to push on the accelerator. You're pulling the nerve root one way. Where's the pain? It's in my big toe. Aha!

I am now going to inform my inspection of the MRI. Because the radiologist missed it. They're not going to find a tarloff cyst distal on the fifth root. But I know that the symptom and the assessment took me there logically to say, I know there's something hanging up there that's directionally specific. It's not a friction, it's a direction-specific tension. There's the tarloff cyst I found.

Now, boy, what's the surgical procedure there? Typically, they'll try and drain the cyst, and it comes right back again, typically. But there's a doc in Dallas who we send all our tarloff cyst patients to, and he has a reasonable rate, at least better than anyone else in dealing with those pesky cysts. A bit of an off-the-wall. I can't do a damn thing about that, a cyst.

It's eroding the bone. They're pesky little things, but here's a surgical referral. Dr. Darrell Bock That's great. Stuart, how often, if you're doing a two-hour assessment on a patient, I assume you're also looking at an MRI. Stuart D. Wals. Let me stop that. After the first year of the experimental research clinic running two years, I changed it to a three hour consult. Wow.

Yeah, I need it even more time. So now if they're an old athlete and they still have films on the film. Remember how we used to get MRIs? I read them on the reader or I put them up on the screen there. So yeah, no, full medical images we go through. What are the things you're looking for in the MRI that maybe aren't as readily apparent? In other words, what are you looking at in an MRI that isn't obvious to the radiologist? Because presumably, yeah, you can maybe explain to somebody what the MRIs are showing, but you're getting axial cuts, you're getting coronal and sagittal cuts, they're T1 weighted, they're T2 weighted, so they highlight the disc, a nice healthy disc looks white on the MRI, of course mine are jet black. What are things you're picking up on that MRI read?

Well, all of the things that you've mentioned, I don't know if you looked at my CV and the number of papers and the topics that we covered over the years, but the very last study that I ever published as a professor was exactly that. We took a cohort of whiplash patients. I didn't do very much cervical spine specific work. Most of mine was lumbar. But just to answer your question, we took whiplash patients. Every single one of them had been denied compensation because they're now more than two years post whiplash. They still continue to have symptoms. The medical

profession and the legal system was declaring them pain magnifiers. They were exacerbating their pain for financial gain. Terrible. The MRs said, there's no reason for your pain. Really? The MR is a static picture. What do you expect? So we took videofluoroscopy, which you know is a real-time moving x-ray. So we're watching the bones move now, and we would have them move through their pain. And their pain wasn't very rarely at the end range of motion. It was actually somewhere in the middle of the range, and they would move their head like this, and then the spine would clunk.

And then they go, oh, and then they continue to move through. On the video fluoroscopy, we watch the rotations occurring between every vertebra, but we know what instability is. It's when the rotation stops and the shear begins. So the ratio of rotation and shear is the marker of that cervical instability. So if I can just show with my hands, here would be the neck moving, rotating well, and then it would clunk. It was the clunk that corresponded 100% with the shot of pain.

Now, you and I both know that when a muscle contracts, it does two things. It creates force, but it also creates stiffness. The body uses stiffness to control motion. Okay, so if you just want to observe me now and you can play along and do this if you like, I want you to lightly stack your ears over your shoulders and have a pitch to your head that's neutral. Stare straight ahead. Now, lightly touch yourself under your jaw, just above your Adam's apple.

Don't retract your two stiff feet. Relax. Now, push your tongue hard to the roof of the mouth behind your front teeth. You felt the deep flexors activate. Now, corners of your mouth grimace down. Do this to your neck. Now, keep that. Imagine the person who's rotating and then has the clunk. Keep that, controlling stiffness, and repeat the

offensive movement. Would you believe in most people the clunk was arrested? It was gone. Proving that the MR had no ability to pick up that dynamic pain trigger. We just proved what their pain trigger was. You can imagine the psychological relief that they had to know that it isn't in their head. The medical profession was wrong. Finally, they're empowered now because they have a strategy to start learning just a little bit of a strategy to take the clunk out. If you arrest the clunk over time, the joint will stiffen. The bad news is you don't move so well through that joint.

The good news is the pain clunk is gone. So we all experienced this and you're going to be experiencing this now over the next 15 years, particularly if you're in your early 50s. Things are going to be stiffening in your body. The good news is your pain will go. You know who really gets this? I've worked with a couple of former Mr. Olympians. That's the top professional level of bodybuilding. They put a lot of mileage on their

joints. They don't really get joint pain when they're competing because the muscles are so big, so bulky, they have enormous wrench handle moment arms and the stiffness holds the joints together. When we work with them, tapering down back to civilian life. Some of them don't look that different than you and me. Believe it or not, what they look like in their former glory, they ain't like hell. All our joints have these shearing translations to them now. So the cure is getting a little bit of the muscle bulk back to add some controlling stiffness and all our aches go away. Anyway, these are all sort of fun stories. I don't know if that's really answered your question on instability, sciatica, brachial plexus nerve traps, numb

thumb and first finger, whatever. They're not indicators for surgery at all. Try some of these voluntary skills and let nature take its course most of the time. And I can with confidence prove it and say most of the time it will work out well with some patients and skill. One of the really good spine surgeons I know and you can always tell a great surgeon by talking to them and maybe I'm fortunate because having trained as a surgeon you sort of learn what the signs are of the hacks and the good ones and as we can all attest to in our own

respective profession we're pretty good at picking up who the good ones are and the bad ones are. But speaking to this spine surgeon, it's just really clear she's a really good surgeon. And one of the signs of a really good surgeon is a surgeon who's really happy to not operate. The really good surgeons are really happy to not operate on somebody. Partly what makes them so good is their judgment. It's their knowing who to operate on and who not to. We did a really fun exercise one day where we went through my MRI.

Every time I get an MRI for another reason, if it's gonna get any sort of back cut, I just send it to her, even if it's not a dedicated spine MRI. And I say, what do you think of this? Does it look any worse? And again, we're always collectively amazed at how bad my spine looks on MRI relative to the fact that I don't have any symptoms.

One of the discussions we had prompted her to contrast my back with that of another patient she had who has no obvious disc pathology and yet is in debilitating pain. And she said, look at the difference and again, I'm not saying this just to be boastful, but I'm just trying to make the contrast. She goes, look at the difference in the musculature of your psoas, your QL, your erector spinae. Like these are big, beefy muscles here. And now compare it to this other patient. First of all, the muscles are about half the size and they look like Wagyu. They're very fatty. And the way she was explaining it to me,

she goes, this is a person who's never lifted anything in their life. And they don't have any of the disc pain. Their discs haven't been decimated like yours have, but they're more debilitated. Their inactivity has led to instability and tremendous pain. You've already sort of alluded to this where we've agreed that the deadlift till you drop strategy and the do nothing strategy are both bad.

But can you speak a little bit to why that person might be in pain? Because what I don't want anybody to come away from this podcast feeling is, oh, I better not lift weights, because that's clearly the wrong message. 100%. Okay, I'm so glad you brought this up. I would love to talk to her and I'd say, tell me about your training program or your daily routine or your life in physical terms. I will bet she's a mobility monster. She keeps pushing the end range, softening the joints even more. So on MRI, they look plump and pristine. I bet if we put her under load or we put her in bed and she had this instability

that I've showed earlier and she lays in bed and the joints just fall like that a little bit, she'll get a hell of an ache to her back. My first question would be, when you roll over in bed, do you ever have a sharp pain? That's a beautiful follow-up question. It is so indicative of if she has nice plump discs, but micro movements. How many pillows do you go to bed with at night? That is a wonderfully telling question. The more the pillows, the more the joint instability. It's quite high-coral. Anyway, I'd love to have that conversation with her, and I will bet we will get some real insight from that. Versus the person who has a mature strength history

and the joints are held together, a little bit of arthritis and people are gonna nail me for this one, but a little bit of arthritis is good for adding certain amount of joint stability and holding it all together. I had a fracture of C4 as a young fella. Oh, I would have some terrible episodes checking my blind spot

or craning my neck to back a trailer up or something. I have zero pain now. My neck is bulletproof again. It looks horrible on a CT or an MR. But my point is the arthritis has now stabilized the joint. All the pain is gone. I don't move it very well, but I don't worry about it. My sister's a vet. She sends me X-rays of a dog. Terrible. And as you know, spine arthritis and nerve compromise in dogs, which is very breed specific as well. They lose their hind end. It just atrophies just like in a person. But anyway, she'll send me this x-ray of the dog. She says, what do you think this dog's doing

right now? And I said, well, it's just laying in its bed. She goes, no, that just won the Frisbee championship. The Frisbee catching championships. So again, I just keep coming back to the assessment. Again, I just keep coming back to the assessment. And between you and I, I don't ever want to see another MRI of my spine until the pattern doesn't fit and I can't move the pain anymore. The time I ever want to see an MRI of my own back. I'm like you, doesn't look so good.

However, I've got a few miles on my back and I'm the person I am today because of that, I do everything I want to do with certain guidelines. I'm not 16 and I don't have infinite capacity. So I play with that tipping point all the time. What would you say to the person who's watching or listening to us right now? And I realize that there's a pretty good chance that by now, because we're a couple hours into this podcast, if you have never experienced back pain, you might not be listening anymore. Because the truth of it is, there's gonna be a lot of people

listening, because if you've experienced back pain, especially if it's happened more than once, or if it lasted more than a week or so, this is a riveting discussion. But if you were talking to a person of any age who had yet to experience it, but in particular, maybe a young person, someone in their 20s or 30s, what would you say to them? And how would you counsel them with respect to what they could do to maximize the longevity

of their spine? What a fabulous question. what they could do to maximize the longevity of their spine. What a fabulous question. If I was to say to you, a young fella comes into your office with a cigarette hanging out of his mouth. What would you say to him that he hasn't already heard? I would love to take you over to the cancer ward

at the hospital, and I wanna show you how your last days are going to look. That might convince a few of them on the lunacy of what they're doing to themselves. It won't be 100% effective, and I would hazard a guess. It wouldn't be close to 100%. Their friends and peer pressure is far more important for them now. That's how I'm going

to answer the question you just asked of me. I don't have very good luck when I see someone who's just all balled up. A kid called me, not a kid, a 30-year-old called me last week. This guy was all balled up like this and he said, ah, he says, whenever I do exercise, I'm just exhausted. I said, oh yeah. I said, would you move away from your desk a little bit and would you ask someone to come in and hold your cell phone up so I can see all of you and there he was. And I said, all right, would you now sit at your stool, your chair, sit upright for me? Do Jeff Payne, and he goes, not. And I said, good. Bop your chest down and slouch and lower your head.

Jeff Payne says, yeah, I do. Now don't you think I just proved to him what caused his pain? He said, well, I've heard that before. I've sat like a cashew and that was his exact words since I was 14. I coached him, okay, sit up, lay on your tummy for a little bit, let this thing calm down. By the way, what do you do when you get up in the morning? Well, I go down and I get a coffee and I said, how do you get to work?

He says, I drive and I said, tomorrow I want you to get up half an hour early and go for a walk and it was snowing here. So I said, it's snowing outside. You live in LA, get your, you know what? Out of bed and go for a walk for half an hour tomorrow morning before you get in your car. Do you know he was bucking me on that? So to your point, I don't think I changed his behavior one little bit and he's going to have to suffer a little bit more before he comes to a realization that he does have the power to do something. And I know your thesis loud and clear in Outlive.

We're identical. We're trying to get people on a program now when he's 30 and not wait to have more misery and more misery. It's so hard to motivate someone. Maybe you have a hint for me. I share your sentiment exactly and that's why I've often referred to that third bout of back pain that I had, the one that lasted for a year as the best worst experience of my life. It was the worst experience in that I wouldn't wish that duration or depth of pain on anyone, but what was so good about it is that it lasted for so long that it created a lifelong change in behavior and an appreciation for something, which is without that experience, this idea of a centenarian decathlon wouldn't exist.

Because you have to sort of see what a life looks like with immobility and pain. Because even though I was only 27, I lived that year as though I was 87. And a year is long enough that it imprints. If it was only a week, no matter how bad it is, I don't think it would have imprinted. But a year of that really imprinted in me. I've said this before many times, but to this day, I still enjoy parking as far away as possible in the parking lot, even if there are plenty of spots close to the grocery store or wherever, because I remember what it was like to not be able to walk from the car to the grocery store. So unfortunately, that's probably the nature of our species in that it's very difficult to

make a short-term sacrifice for a long-term objective without a more pressing reason. So instead, I'll turn my attention to who I think is the larger population listening to us, which are the people who have experienced either personally or through watching someone they care about perhaps. Let's start with this. What are the best online resources we can point people to that can help with the types of exercises, maybe some do's and don'ts around lower back pain? I love that you even clarified around the big three, which is, hey, the big three are great if you need stability, but if you need mobility, we might need some different exercise.

So how can people sort of navigate their way through that? I challenged myself with exactly the same issue 15 years ago, just as the internet was getting going. But here's the thing. There is no such thing as non-specific back pain. And if that's what the person operates on in their strategy, this non-specific thing, it will only be dumb luck if they're able to come up with a strategy to mitigate it. They have to have an assessment.

Well, they can go and see someone who is very knowledgeable in converging on an understanding of their pain most of the time. Well, short of that, I wrote Back Mechanic. Now, it's not on the internet. And the reason is they have to have some background understanding of how their back works and then go through a series of self-tests. That's what the book does. The first thing is it just says, draw a table. What are activities that cause you pain? What are activities that either take your pain away or are neutral? Write them all out. Now, here is how you, I term, recognize those. All of those activities involve you bending backwards. Guess what? Change a light bulb overhead

that triggers your pain. We're starting to learn a little bit about what could the candidates be. Then we take them through some physical tests. Sit on a chair, slouch, extend, drop one shoulder back, hold five pounds out at front with arms straight. So that's a compression test. Then we do a few self-shear tests. Then we do some nerve-tensioning postures to start converging on subcategories of their pain. Then we say, if you have this subcategory, let's do a real simple one. You get pain when you sit in front of your computer

going for a walk is relieving. The next person sitting in the computer is their relief and they go for a walk and that causes their pain. Probably more in the stenosis older person kind of category. The other one is a younger dynamic disc bulge. Okay, sit with a lumbar support. Number one. Number two, we are now going to have a strategic exercise session. You're going to do it every day, you're going to do the big three, we'll mobilize the hips, you're not going to sit longer than an hour at your computer, you just cannot reach a stage of sufficient health if you continue with that behavior, etc. So that's why you won't find it on the internet.

You're going to find a lot of people who do not have the expertise. Oh, here's the quick fix for your back pain. Well, good luck with that. So that's my answer to your question and the solution. Just going back to listening to you as you started to answer that question, I've got a little bit of good news for you in terms of your own back. By the way, I know who I'm talking to, so I know you get this, but this is for the listenership. I retired early.

I retired when I was 60. I reached a stage where I realized what my job was. I started as a professor in 1986. Student meetings meant students came to see you and we would get up and we'd work through things and we'd do things in the laboratory and whatnot. And then the students started to migrate to this idea, oh sir, could we have an online call for student hours? No, you can't.

You get down here and we're going to work through this problem. In other words, my job got turned into a sitting job and it was killing me. And I realized that my health was declining, my fitness was declining. I still walked to the university. I strategically bought a home right on the edge of campus. So I would have a 20 minute walk to and from my office and laboratory.

Still, I was declining. So I walked away. I shut the door in my office. I said to the graduate students, there's all my books. Go take them. To all the other professors, there's my lab. Go take it. And I just walked away, never thinking that anyone would ever ask me again because I'm

not producing new data anymore. I was sort of wrong on that estimate. But anyway, my point is, Peter, I'm healthier now than I ever was in the latter 15 years of my computerized work life. I hardly go on the computer. It's fabulous. I can talk about my life now if you want and what I do. But my point in this story is, I think you're going to look forward to a resurgence of your health. Maybe you've got it dialed in with your seeing patients and traveling and everything else. Maybe you don't. But trust me, when you retire, and that doesn't mean leaving your whole medical family and expertise. I mean, I'm sort of working right now. I still see patients two days a week. It's a wonderful marker for my week.

I love it. But the other five days, I live a healthy life. Anyway, my point in all of that is things are going to get really better for you. They're not a decline more. I've heard you say that. And I think, come over with me, man. Spend a couple of days and you'll see how you're not on this decline as you think is a fated complete.

You just said something a moment ago that I was going to ask you about. So, at the risk of overwhelming you, because I know that there are going to be so many people listening to us who are going to say, you know what, I am not happy with the assessment or lack thereof that I've received. I'm not happy with the care that I'm receiving with respect to my lower back injury. I need to go and see Dr. McGill. What is involved in arranging that type of a consultation with you? I feel awkward saying this, but that's why I wrote Back Mechanic. So, I don't see anybody until they've read the book. Most of them say, I don't need to see you now. So, they've been through the self-assessment,

they've got enough out of it. Now, if they're not getting enough out of it, on our website, backfitpro.com, we have two layers of clinicians. We have the com, we have two layers of clinicians. We have the certified clinicians who've taken that 50-hour course. They've gone through the hands-on skills training. They've written the exam, but I've never worked with them personally, but they're all there on a page. Then we have a different level called master clinicians. I have worked with every single one of those people and trained them. I've seen patients with them. They have my confidence now that I can send them any patient and they will subcategorize them and know pretty well what to do with them. I continue to train those individuals. I seek out stars or people who have the passion and the skill and I go to them and say, would you now study with me and I'd like you to become one of our master clinicians.

Darrell Bock How many master clinicians are there in North America, Stuart? Stuart Sturgeon Not many. I don't know. A dozen, 15 maybe, something like that. Darrell Bock But they're all identifiable on the website, which was backfitpro.com. Correct. And the certified, that's growing all the time. There's maybe 30 or 40 of them. We add to that every couple of months.

Okay. I think people in reading that book, it's quite a quick read. It was a very difficult book to write, as you can imagine. I've written my medical textbooks for my medical colleagues. Those are easy to write, as you can imagine. I've written my medical textbooks for my medical colleagues. Those are easy to write. You put in the references, you make your points, you show the strength of evidence, et cetera. But you can't do that with the public. You have to give them enough of the truth to guide a effective strategy, but you can't overwhelm them with jargon and all of that. So that's why those things are so difficult to write, but people tell me that back mechanic

in any case, I sent you a copy. I hope you got it. And not only got it, I greatly appreciated the inscription in it. Thank you. Oh, yeah. Okay. That was special. Okay, that was special. Heartfelt. In any case, that is my solution to that conundrum. And that's why going to the internet, as you know, it's the Wild West. You can get screwed up as much as you can be helped. Well, Stuart, this has been a really enlightening discussion for me and given how much I've

thought about this topic. I think that says something, but it tells me that more than anything else, a lot of people listening to this, which again, I think is a lot of people who can relate to what we're talking about personally. I think this, I hope offers more than just a glimmer of hope and also a set of resources that people can look to. And I will take you up on this offer. The next time I'm in Toronto, we'll make that trip up to Gravenhurst. Apologies for my poor Canadian geography. I always thought Gravenhurst was just outside of Toronto.

I didn't realize it was that far north. Yeah. Huntsville Brace Bridge, Gravenhurst, if you know that area, right in the heart of Muskoca. Yeah. So just from like Aurora, we're talking like what, 90 minutes, two hours? No, about an hour and a half north of Aurora.

Okay. Yeah. All right. Well, we'll make that happen. Okay. Well, I hope so. Peter, I've looked forward to this day ever since we scheduled it a couple of months ago. The leadership that you've provided is fabulous. I've spent many hours listening to your podcasts and getting wisdom from your guests and the level that you take all these issues to is just the foundation I need for a lot of the things that I think about. For all you do, thank you so much. The way you posed your questions today

were not really typical, so I appreciate that very much. But again, thanks for all you do. Well, thank you for what you do, because that's where I'm learning today. So thank you, Stuart. Okay, my pleasure. Thank you for listening to this week's episode of The Drive. It's extremely important to me to provide all of this content without relying on paid

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